Fibromyalgia is a condition characterised by muscle aches, stiffness, and tender points in specific areas. People effected by this disorder are also known to have sleep difficulties, morning stiffness, and fatigue. There have been many ideas as to why this disease starts and the way it's maintained in the body, however there may still be much understand. Regardless of this, there may now be good proof to back up the concept that fibromyalgia is a situation caused by the central nervous system (spinal column and mind). The central nervous system is likely processing pain in a dysfunctional way. Pain science research has shown that we have to look at the major changes that happen within the central nervous system in an effort to be more practical in managing painful conditions such as fibromyalgia.

Just like other persisting pains, there are often a myriad of other symptoms which might be noticed, a few of which may make little to no sense in context. One of the most necessary components of managing and treating persistent pains is to understand problem as well as the symptoms. 'Explaining' pain is fundamental in dealing with fears and answering questions that are causing fears and anxieties which alone can amplify these pains. Understanding the influences of the pain and the components that can aggravate this pain will help you manage the pain and find effective therapies and treatments. This has been evident by a examination which included pain training on a selected test group that is generally used in the clinic. It was discovered that the test group showed an incredible improvement after they had taken a class to help them better understand pain science.

There have been many studies which have used mind scanning equipment to look at mind activity during physically painful experiences. The findings support new theories on pain which are known as the 'pain neuromatrix'. This was all developed by Ron Melzack. This theory describes a community of nerves in the brain that only when lit up in a specific way would create the acutely aware experience of pain. The areas of the brain that form this matrix are accountable for control of motion, sensation, anxiousness, reminiscence, focus, emotion, belief, planning and vision to name a few. Certainly one of these areas when activated can set off the opposing areas by way of interconnections, thereby causing some activity in another part of the brain throughout the network. For example, seeing somebody bending can provoke your back pain from observing the motion and stimulating activity in different elements of the matrix. We now have a 'neurosignature' for all activities and ideas, this being a pattern of activity inside a community of neurons that's responsible for our experience, for example walking, talking and writing. One area that's particularly worth pointing out is the amygdala which is used for anxiety. By means of its connections with different components of this neuromatrix the amygdala can set off the neurosignature for pain. This means that if you're feeling fearful or anxious because of not understanding your symptoms of fibromyalgia for instance, the ache could be increased.

Pain is a standard sensory experience that is a part of a response created by the brain (not the imagination!) to encourage some action. This is the same as thirst or starvation that motivates the action of searching for food or water for survival. Different parts of this response embody reflexive motion, changes in blood circulation and immune activity to create healing. Usually the tissue will heal after being broken and the sensitivity that develops usually will go away. Typically this sensitivity does not occur and instead persists. Which means the tissues heals however the nervous system remains in an excitable state, sending warning messages even though there is no such danger. Clearly the management of this sort of problem is different and requires a very broad lens to consider the influences upon the pain experience in fibromyalgia. With a chronic condition the therapy must concentrate on the activity of the central nervous system and the way by which it processes the information related to pain. Normal motion is painful because the processing of the information leads to the brain believing there to be a threat to the tissues. We know that the tissues are actually not unhealthy however the nerves that are inside tissues are excitable and the central nervous system is sensitive and that mean that ordinary contact and movements are perceived to be 'harmful'. Altering the mind's notion of this requires more than tissue focused treatment is providing. Instead we need interventions such as education, training, coping skills, learning to relax and tactile discrimination .

Science clearly tells us that pain isn't a reliable indicator of tissue damage. Some examples include phantom limb pain and some of the kinds of pain-free traumas. This suggests that the mind should prioritize the data from the tissues in the importance of survival, and decide what motion is most important. This fact is significant in understanding and managing pain symptoms of fibromyalgia in an efficient way. Suffering with the symptoms of fibromyalgia typically means widespread pain, stiffness, tenderness within the tissues, fatigue and temper changes. The activity in the central nervous system is the reason for many of these symptoms and it is vital that this is understood if you suffer from fibromyalga. Linked with the pain processing pathways in the brain are the activities of the immune system and stress physiology. Which means if you are feeling unwell the pain may be amplified by the elevated release of immune chemical compounds that sensitise the nervous system. The cascade of hormones released when the body deals with stress (pain is a stressor with fibromyalga) can even amplify the pain and be reason for stiffness, particularly in the morning. There was a lot work that went into trying to understand the links between common health, sleep, mood and pain. Fibromyalgia is a complex disease with numerous contributing factors. The understanding of the causes and pathology of the disease has increased significantly however. But there is still much to be learned.